Decompression Therapy

Decompression Therapy

Incorrect and Correct Lifting

Standing

Spinal Decompression Therapy utilizes a unique equipment designed to gently and effectively reduce disc bulges and disc herniations as well as increase spinal mobility. By applying a targeted pull to the spine with specific angles and calculated weight, spinal decompression therapy causes a 1-2 mm opening of the involved disc. This opening allows for a "vacuum" effect to happen to the bulging/herniated material, and effectively pulls the material back away from the irritated nerve leading to pain relief. Spinal decompression is not traction which has poorer results.

Traction, Physical Therapy, Manual Manipulation

The computerized traction head on the decompression table is the key to the therapy's effectiveness. The preprogrammed patterns for ramping up and down the amount of axial distraction eliminate muscle guarding and permit decompression to occur at the disc level. This creates a negative pressure within the disc, allowing the protruded or herniated portion to be pulled back within the normal confines of the disc, which permits healing to occur.

Spinal decompression also allows vital nutrients and blood supply to rush into the disc which helps speed the heeling process. This, combined with our phototherapy, helps to increase healing of the area by over 80% faster.

Low back pain has become an epidemic. It is responsible for the 2nd highest number of visits to the emergency room. Many of these cases are due to bulging/herniated discs. Surgery is the option for many.

Those suffering from spinal pain are generally in pain for many years and have tried conventional remedies including medication, physical therapy, chiropractic, acupuncture or home exercises.

If you suffer from spinal pain and have not had success with other treatment, then the new state-of-the-art spinal decompression therapy is for you or your loved one experiencing:

  • Disc bulging/herniation
  • Sciatica
  • Spinal stenosis
  • Degenerative disc disease
  • Radicular arm pain
Spinal Decompression

Below is the clinical research on spinal decompression. The results are phenomenal.

Spinal Decompression Clinical Research

By Thomas A. Gionis, MD, JD, MBA, MHA, FICS, FRCS, and Eric Groteke, DC, CCIC

The outcome of a clinical study evaluating the effect of nonsurgical intervention on symptoms of spine patients with herniated and degenerative disc disease is presented.

This clinical outcomes study was performed to evaluate the effect of spinal decompression on symptoms and physical findings of patients with herniated and degenerative disc disease. Results showed that 86% of the 219 patients who completed the therapy reported immediate resolution of symptoms, while 84% remained pain-free 90 days post-treatment. Physical examination findings showed improvement in 92% of the 219 patients, and remained intact in 89% of these patients 90 days after treatment. This study shows that disc disease—the most common cause of back pain, which costs the American health care system more than $50 billion annually—can be cost-effectively treated using spinal decompression. The cost for successful non-surgical therapy is less than a tenth of that for surgery. These results show that biotechnological advances of spinal decompression reveal promising results for the future of effective management of patients with disc herniation and degenerative disc diseases. Long-term outcome studies are needed to determine if non-surgical treatment prevents later surgery, or merely delays it.

RESULTS

According to the self-rated Oswestry Pain Scale, treatment was successful in 86% of the 219 patients included in this study (Table 2, page 39). Treatment success was defined by a reduction in pain to 0 or 1 on the pain scale. The perception of pain was none 0 to occasional 1 without any further need for medication or treatment in 188 patients. These patients reported complete resolution of pain, lumbar range of motion was normalized, and there was recovery of any sensory or motor loss. The remaining 31 patients reported significant pain and disability, despite some improvement in their overall pain and disability score.

Diagnosis MRI
Findings
No. of CaseS Female Patients Male Patients Positive Result No Result % of Success
Single Herniation Lateral 67 26 41 63 4 94
Single Herniation Central 22 11 11 20 2 90
Single Herniation w/ Degeneration 24 5 19 24 0 100
Single Herniation Lateral w/ Degeneration 32 14 18 29 3 91
Multiple Herniations w/o Degeneration 57 21 36 39 18 68
Multiple Herniations w/ Degeneration 17 2 15 13 4 77
TOTAL 219 79 140 188 31 86

Table 2. Results on self-rated Oswestry Pain Scale after treatment.

In this study, only patients diagnosed with herniated and degenerative discs with at least a 4-week onset were eligible. Each patient's diagnosis was confirmed by MRI findings. All selected patients reported 3 to 5 on the pain scale with radiating neuritis into the lower extremities. By the second week of treatment, 77% of patients had a greater than 50% resolution of low back pain. Subsequent orthopedic examinations demonstrated that an increase in spinal range of motion directly correlated with an improvement in straight leg raises and reflex response. Table 2 shows a summary of the subjective findings obtained during this study by category and total results post treatment. After 90 days, only five patients (2%) were found to have relapsed from the initial treatment program.

Diagnosis MRI Findings Improved Gait Sluggish to Normal Reflexes Improved Sensory Reception Improved Motor Limitation Abnormal to Normal Straight Leg Raise Test Improved Spinal Range of Motion
Single Herniation Lateral 98% 98% 96% 90% 92% 95%
Single Herniation Central 100% 100% 94% 92% 96% 90%
Single Herniation w/ Degeneration 99% 96% 90% 84% 94% 90%
Single Herniation Lateral w/ Degeneration 94% 97% 94% 88% 90% 92%
Multiple Herniations w/o Degeneration 96% 94% 94% 81% 82% 92%
Multiple Herniations w/ Degeneration 92% 94% 88% 82% 80% 82%
AVERAGE IMPROVEMENT 96% 96% 93% 86% 89% 90%

Table 3. Percentage of patients that had improved physical exam findings post treatment.

Ninety-two percent of patients with abnormal physical findings improved post-treatment. Ninety days later only 3% of these patients had abnormal findings. Table 3 summarizes the percentage of patients that showed improvement in physician examination findings testing both motor and sensory system function after treatment. Gait improved in 96% of the individuals who started with an abnormal gait, while 96% of those with sluggish reflexes normalized. Sensory perception improved in 93% of the patients, motor limitation diminished in 86%, 89% had a normal straight leg raise test who initially tested abnormal, and 90% showed improvement in their spinal range of motion.

SUMMARY

In conclusion, nonsurgical spinal decompression provides a method for physicians to properly apply and direct the decompressive force necessary to effectively treat discogenic disease. With the biotechnological advances of spinal decompression, symptoms were restored by subjective report in 86% of patients previously thought to be surgical candidates and mechanical function was restored in 92% using objective data. Ninety days after treatment only 2% reported pain and 3% relapsed, by physical examination exhibiting motor limitations and decreased spinal range of motion. Our results indicate that in treating 219 patients with MRI-documented disc herniation and degenerative disc diseases, treatment was successful as defined by: pain reduction; reduction in use of pain medications; normalization of range of motion, reflex, and gait; and recovery of sensory or motor loss. Biotechnological advances of spinal decompression indeed reveal promising results for the future of effective management of patients with disc herniation and degenerative disc diseases. The cost for successful nonsurgical therapy is less than a tenth of that for surgery. Long-term outcome studies are needed to determine if nonsurgical treatment prevents later surgery or merely delays it.

Thomas A. Gionis, MD, JD, MBA, MHA, FICS, FRCS, is chairman of the American Board of Healthcare Law and Medicine, Chicago; a diplomat professor of surgery, American Academy of Neurological and Orthopedic Surgeons; and a fellow of the International College of Surgeons and the Royal College of Surgeons.

Eric Groteke, DC, CCIC, is a chiropractor and is certified in manipulation under anesthesia. He is also a chiropractic insurance consultant, a certified independent chiropractic examiner, and a certified chiropractic insurance consultant. Groteke maintains chiropractic centers in northeastern Pennsylvania, in Stroudsburg, Scranton, and Wilkes-Barre

REFERENCES

  1. Eyerman E. MRI evidence of mechanical reduction and repair of the torn annulus disc. International Society of Neuroradiologists; October 1998; Orlando.
  2. Narayan P, Morris IM. A preliminary audit of the management of acute low back pain in the Kettering District. Br J Rheumatol. 1995;34:693-694.
  3. McDevitt C. Proteoglycans of the intervertebral disc. In: Gosh, P, ed. The Biology of the Intervertebral Disc. Boca Raton, Fla: CRC Press; 1988:151-170.
  4. Bogduk N, Twomey L. Clinical Anatomy of the Lumbar Spine. New York: Churchill Livingstone; 1991.
  5. Cox JM. Low Back Pain: Mechanism, Diagnosis, and Treatment. 5th ed. Baltimore: Williams & Wilkins; 1990:69-70, 144.
  6. Cyriax JH. Textbook of Orthopaedic Medicine: Diagnosis of Soft Tissue Lesions. Vol 1. 8th ed. London: Balliere Tindall; 1982.
  7. Nachemson AL. The lumbar spine, an orthopaedic challenge. Spine. 1976;1(1):59-69.
  8. Ramos G, Martin W. Effects of vertebral axial decompression on intradiscal pressure. J Neurosurgery. 1994;81:350-353.
  9. Shealy CN, Leroy P. New concepts in back pain management: decompression, reduction, and stabilization. In: Weiner R, ed. Pain Management: A Practical Guide for Clinicians. Boca Raton, Fla: St Lucie Press; 1998:239-257.
  10. Pal B, Mangion P, Hossain MA, et al. A controlled trial of continuous lumbar traction in back pain and sciatica. Br J Rheumatol. 1986;25:181-183.
  11. Weber H. Traction therapy in sciatica due to disc prolapse. J Oslo City Hosp. 1973;23(10):167-176.
  12. Yong-Hing K, Kirkaldy-Willis WH. The pathophysiology of degenerative disease of the lumbar spine. Orthop Clin North Am. 1983;14:501-503.
  13. Matthews J. The effects of spinal traction. Physiotherapy. 1972;58:64-66.
  14. Goldfish G. Lumbar traction. In: Tollison CD, Kriegel M, eds. Inter-
  15. disciplinary Rehabilitation of Low Back Pain. Baltimore: Williams & Wilkins; 1989.
  16. Onel D, Tuzlaci M, Sari H, Demir K. Computed tomographic investigation of the effect of traction on lumbar disc herniations. Spine. 1989; 14(1):82-90.